Talking to Your Child About Weight: A Family Approach

On By Dr. Scott RennieIn Weight Loss & Metabolic HealthLeave a comment

Pediatric obesity affects nearly one in five children in the United States (Stierman B, Afful J, Carroll MD, et al. National Health Statistics Reports, No. 158, 2021). In primary care, we’re often the first to see the signs. We see families regularly. That gives us a real chance to guide prevention and help establish healthy habits early.

The American Academy of Pediatrics recommends eleven visits in the first two years of life. Those visits do more than cover vaccines and ear checks: they’re a chance to track growth, notice concerning patterns early, and build trust with parents. Short counseling moments, repeated over time, often make more impact than a single long lecture. Small doses, consistently given.

The Chronic Care Model gives us a structure to work from. It starts with decision support, like using BMI and growth charts to flag risk. It builds in self-management tools such as plate planners that make meal discussions concrete, emphasizes delivery system design, and connects practices to community resources so families don’t feel like they’re managing this on their own (Dietz WH, Lee J, Wechsler H, Malepati S, Sherry B. Health Affairs. 2007, 26(2):430-440).

During pregnancy, maternal smoking and psychological stress both raise a child’s later obesity risk (Rayfield S, Plugge E. J Epidemiol Community Health. 2017, 71:162-173) (Dancause KN, Laplante DP, Oremus C, Fraser S, Brunet A, King S. Pediatr Res. 2012, 71:126-131). From birth through the toddler years, setting expectations around normal growth and picky eating helps prevent overfeeding. In preschool and elementary school, reinforcing habits around meals and activity can keep BMI from drifting up. Once kids reach adolescence, weight management often needs more direct intervention (Cardel MI, Atkinson MA, Taveras EM, Holm JC, Kelly AS. JAMA Pediatr. 2020, 174:609-617).

Parental obesity, maternal smoking during pregnancy, a chaotic home feeding environment: all of it raises the odds of rapid early weight gain (Stettler N, Zemel BS, Kumanyika S, Stallings VA. Pediatrics. 2002, 109:194-199). These are the kids who benefit most from structured follow-up.

Meta-analyses show up to a 22 percent reduction in obesity risk from breastfeeding, though the effect drops to about 7 to 10 percent once you adjust for maternal obesity and socioeconomic status (Owen CG, Martin RM, Whincup PH, Smith GD, Cook DG. Pediatrics. 2005, 115:1367-1377) (Harder T, Bergmann R, Kallischnigg G, Plagemann A. Am J Epidemiol. 2005, 162:397-403). Breastfed infants are better at regulating their own intake, and breastmilk itself carries bioactive compounds that affect metabolism (Arenz S, Rückerl R, Koletzko B, von Kries R. Int J Obes (Lond). 2004, 28:1247-1256).

I often tell parents to expect a “three-day eating cycle” in toddlers. One day they eat well, the next they pick at food, the third day they do something in between. Framing it this way reassures parents and heads off unnecessary pressure at mealtime. Another helpful concept is covert restriction, simply not bringing high-calorie snacks into the house in the first place. That avoids turning junk food into a “forbidden fruit” while still shaping healthier choices (Ogden J, Reynolds R, Smith A. Appetite. 2006, 47:100-106).

Motivational interviewing works by asking how they view their child’s weight, showing growth charts, and framing recommendations without blame: all of it makes these conversations more effective (Barlow SE. Pediatrics. 2007, 120 Suppl 4:S164-192). Simple, achievable goals work better than long lists. I often start with two diet-related changes, like reducing sugary drinks and adding vegetables, and one activity change, such as more outdoor play.

Checking in every three months, celebrating small wins, reinforcing progress: that’s what keeps families engaged. Counseling doesn’t need to be perfect. It needs to be consistent.

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources:

How Obesity in Pregnancy Affects the Baby and Placenta

On By Dr. Scott RennieIn Menopause & Women's Health, Weight Loss & Metabolic HealthLeave a comment

As healthcare providers, we’ve long recognized the importance of maternal health in shaping pregnancy outcomes. What’s become clearer in recent years is the role of epigenetics, changes in gene expression that don’t alter DNA itself but influence how genes are turned on or off.

When we look at pregnancies complicated by obesity, DNA methylation patterns in the placenta are altered, with increased methylation suppressing genes that matter for nutrient transport and fetal growth. DNA hydroxymethylation, usually tied to active gene expression, tends to drop at the same time. One study found a 21 percent increase in methylated regions and a 31 percent decrease in hydroxymethylated regions in placentas from obese pregnancies compared with lean ones (Mitsuya K, Parker AN, Liu L, Ruan J, Vissers MCM, Myatt L. PLOS ONE. 2017, 12(10):e0186115).

These changes show up directly in the structure and function of the placenta. Inflammatory changes are common, disrupting hormonal signals and communication between maternal and fetal systems, while lipid buildup in placental cells interferes with normal function and impaired blood vessel development slows villous maturation, reducing the placenta’s ability to exchange oxygen and nutrients efficiently (Saben J, Lindsey F, Zhong Y, et al. Placenta. 2014, 35:171-177).

Mitochondrial function also suffers. Placental cells from obese pregnancies generate less ATP through oxidative phosphorylation. Less energy available means nutrient transport and other vital processes get compromised, which can directly affect fetal growth and development (Mele J, Muralimanoharan S, Maloyan A, Myatt L. Am J Physiol Endocrinol Metab. 2014, 307:E419-E425).

The clinical consequences are real. Growth restriction at one extreme, excessive growth at the other. Both ends of that spectrum carry a higher risk of metabolic problems later in life, including insulin resistance and obesity (Ornoy A. Reprod Toxicol. 2011, 32:205-212).

In practice, weight and metabolic health before and during pregnancy matter more than we once realized. Supporting women to reach a healthier weight before conception can reduce risks. During pregnancy, balanced diets built around whole foods, healthy fats, and complex carbohydrates can help improve metabolic stability. For women with metabolic dysfunction, medications such as metformin may play a role in improving insulin sensitivity.

Regular follow-up lets us track maternal health and fetal growth, and adjust care as needed. The goal reaches past the pregnancy itself. Reducing the child’s long-term risk of obesity and metabolic disease matters just as much.

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources:

  • 1. Mitsuya K, Parker AN, Liu L, Ruan J, Vissers MCM, Myatt L. Alterations in the placental methylome with maternal obesity and evidence for metabolic regulation. PLOS ONE. 2017;12(10):e0186115. https://pubmed.ncbi.nlm.nih.gov/29045485/
  • 2. Saben J, Lindsey F, Zhong Y, et al. Maternal obesity is associated with a lipotoxic placental environment. Placenta. 2014;35(3):171-177. https://pubmed.ncbi.nlm.nih.gov/24484739/
  • 3. Mele J, Muralimanoharan S, Maloyan A, Myatt L. Impaired mitochondrial function in human placenta with increased maternal adiposity. Am J Physiol Endocrinol Metab. 2014;307(5):E419-E425. https://pubmed.ncbi.nlm.nih.gov/25028397/
  • 4. Ornoy A. Prenatal origin of obesity and their complications: gestational diabetes, maternal overweight and the paradoxical effects of fetal growth restriction and macrosomia. Reprod Toxicol. 2011;32(2):205-212. https://pubmed.ncbi.nlm.nih.gov/21620955/

Why Your Body Fights to Regain the Weight You Lost

On By Dr. Scott RennieIn Weight Loss & Metabolic HealthLeave a comment

As physicians, we often see patients who manage to lose weight but can’t seem to keep it off. One of the biggest physiological reasons is adaptive thermogenesis. Understanding what it is, and how the brain adapts too, helps explain why long-term maintenance is so hard.

Adaptive thermogenesis is the body’s way of conserving energy after weight loss. Drop a significant amount of weight, and the body needs fewer calories to function. That makes it harder to hold on to the new weight. The research goes back decades and shows the effect clearly: people who lose ten percent or more of their body weight need roughly 300 to 400 fewer calories a day to maintain that weight than someone at the same size who never lost it (Leibel et al., NEJM, 1995; Rosenbaum et al., Am J Physiol, 2003).

It’s easiest to picture with two patients. Same height, same weight, same activity. One has always been at that weight. The other lost ten percent to get there. The second patient has to eat several hundred calories less, or burn that much more through activity, just to stay even. That’s the metabolic disadvantage weight-reduced patients live with.

What makes this harder is that adaptive thermogenesis doesn’t fade away. Studies show the effect can last for years, even after weight stays stable (Sumithran et al., NEJM, 2011; Rosenbaum et al., JCI, 2008; Martins et al., AJCN, 2020). This isn’t a short-lived adjustment. Patients have to adjust to the reality that the body keeps pushing back against weight maintenance, sometimes for years after the loss.

The brain changes too. Imaging studies show heightened activity in reward and decision-making regions like the globus pallidus, insula, and ventral striatum after weight loss. Food looks more appealing. Cravings get stronger, and resisting them gets harder. Meanwhile regions tied to satiety and control, the hypothalamus and cingulate gyrus among them, go quiet. Patients feel less full, notice their intake less, and have less control over eating. It’s a setup for regain.

Despite these challenges, there are lessons from people who succeed. The National Weight Control Registry has followed thousands of long-term maintainers. On average they eat about 100 to 150 fewer calories daily than peers at the same weight who never lost it. They move more, roughly 45 extra minutes of activity a day. They watch less television, and they show consistent awareness and restraint around eating. Nothing flashy. It just works against the biology.

Exercise, aerobic and resistance both, raises energy expenditure and helps preserve muscle mass, which supports metabolism. Mindful eating and careful tracking help patients avoid unintentional increases in intake. Structured programs and social support provide accountability, which matters when the body is working against the patient. Regular follow-up visits help sustain those behaviors.

Adaptive thermogenesis and brain adaptations stack the deck against weight maintenance. Understanding the physiology makes it easier to set realistic expectations, though, and to design strategies that work with a patient’s biology instead of against it.

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

References:

  • 1. Leibel RL, Rosenbaum M, Hirsch J. Changes in Energy Expenditure Resulting from Altered Body Weight. N Engl J Med. 1995;332:621-628. https://pubmed.ncbi.nlm.nih.gov/7632212/
  • 2. Sumithran P, Prendergast LA, Delbridge E, et al. Long-Term Persistence of Hormonal Adaptations to Weight Loss. N Engl J Med. 2011;365:1597-1604. https://pubmed.ncbi.nlm.nih.gov/22029981/
  • 3. Rosenbaum M, et al. Effects of experimental weight perturbation on skeletal muscle work efficiency in human subjects. Am J Physiol Regul Integr Comp Physiol. 2003;285:R183-R192. https://pubmed.ncbi.nlm.nih.gov/12609816/
  • 4. Rosenbaum M, et al. Leptin reverses weight loss-induced changes in regional neural activity responses to visual food stimuli. J Clin Invest. 2008;118:2583-2591. https://pubmed.ncbi.nlm.nih.gov/18568078/
  • 5. Martins C, Roekenes J, Salamati S, Gower BA, Hunter GR. Metabolic adaptation is an illusion, only present when participants are in negative energy balance. Am J Clin Nutr. 2020;112:1212-1218. https://pubmed.ncbi.nlm.nih.gov/32844188/

Why Exercise Matters for Obesity Beyond Weight Loss

On By Dr. Scott RennieIn Weight Loss & Metabolic HealthLeave a comment

When we counsel patients about obesity management, one of the most common misconceptions is that exercise by itself is the best way to lose weight. On video visits, patients often tell me they’ve started walking every day or signed up for the gym, expecting big changes in their weight within weeks. Sometimes even clinicians lean into that belief. But the data consistently show that diet has a much stronger impact on weight loss than exercise alone.

That doesn’t mean physical activity isn’t important. Far from it. The average weight loss from exercise alone is modest, usually around two to three kilograms. Add diet changes, and outcomes improve significantly. Patients who combine both tend to lose more weight and, more importantly, keep it off better.

The role of exercise extends well beyond weight loss itself. It preserves lean body mass, lowers blood pressure, improves cholesterol, and supports long-term physical function. Pharmacotherapy shows the same pattern: medications can drive the weight loss, but adding structured activity makes the results more sustainable.

The type of activity matters. Aerobic exercise, walking, cycling, swimming, drives calorie expenditure, but resistance training helps maintain or build muscle, which matters most when calories are reduced. The combination works best. Most recommendations center around 150 minutes of moderate-intensity activity per week, which can mean brisk walking 30 minutes a day, five days a week. Some patients prefer high-intensity interval training because it takes less time, though not everyone tolerates it well. For beginners, discomfort and injury risk run higher, so starting gradually makes sense.

It’s important to manage expectations. When a patient logs on discouraged because an exercise program hasn’t led to major weight loss, that’s a teaching moment. Diet has to be part of the plan too, and exercise alone was never going to get there. Once patients understand that, they’re more willing to combine strategies instead of giving up.

Adherence and enjoyment are often the deciding factors. People stay consistent when they choose activities they actually like. One patient may thrive on group classes, another prefers solitary walks with a podcast. Both approaches work, if they’re done regularly.

Daily habits outside the gym matter too. Non-exercise activity thermogenesis, or NEAT, is the energy used in everyday movement: standing during phone calls, walking instead of driving short distances, taking the stairs instead of the elevator, plus gardening, housework, walking a dog, small things patients can relate to. Increasing NEAT can meaningfully raise daily calorie burn without feeling like a formal workout.

For us as providers, diet drives most of the weight loss, but physical activity is essential for maintaining it and for overall health. Framing it this way helps patients set realistic expectations while reinforcing that movement stays a regular part of life.

Reference:

Swift DL, McGee JE, Earnest CP, Carlisle E, Nygard M, Johannsen NM. The Effects of Exercise and Physical Activity on Weight Loss and Maintenance. Prog Cardiovasc Dis. 2018;61(2):206-213. https://pubmed.ncbi.nlm.nih.gov/30003901/

Scott Rennie, D.O.

Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Does Meal Timing Affect Your Metabolism and Health?

On By Dr. Scott RennieIn Weight Loss & Metabolic HealthLeave a comment

When you eat can matter as much as what you eat. Not a fad, a measurable physiological pattern, and one most people never think to adjust. Timing affects weight management, metabolic health, and even the risk of chronic disease, and the field studying the connection between circadian rhythms and eating patterns has a name: chrononutrition.

Typical eating habits in the U.S. don’t line up well with what the research suggests is healthiest. Most people eat less than a quarter of their daily calories before noon, and more than a third after 6 p.m. The average eating window stretches nearly fifteen hours a day (Gill and Panda, 2015). That long, backloaded pattern is linked to higher body fat, worse glucose tolerance, and poorer metabolic outcomes.

Evidence favors eating earlier. In one trial, people who made breakfast their largest meal had better glucose and insulin control, felt more satisfied during the day, and lost more weight than those who made dinner the main meal (Jakubowicz et al., 2013). That tracks with circadian biology: the body handles nutrients more efficiently in the morning than late at night.

Breakfast does more than supply calories. Regular breakfast eaters tend to take in more vitamins and minerals overall (St-Onge et al., 2017), and they show lower rates of obesity and more stable weight over time. Skipping breakfast, by contrast, tracks with higher BMI, larger blood sugar swings, and greater cardiovascular risk.

Time-restricted eating fits the same pattern. Narrow the eating window to eight or twelve hours and total calorie intake often falls on its own, insulin sensitivity improves, and fat oxidation increases (Gill and Panda, 2015). Early time-restricted eating, where meals cluster in the morning and early afternoon, improves appetite control and fat metabolism specifically (Ravussin et al., 2019).

Intermittent fasting runs on the same logic. The most common version is 16:8: fast sixteen hours, eat during an eight-hour window. Alternate-day fasting has also been studied, and it produces weight loss similar to plain calorie restriction, though dropout rates run higher (Trepanowski et al., 2017).

What the science of chrononutrition shows is that timing matters. Shifting more calories to the first part of the day improves metabolism and weight control, breakfast and early meals set the body up for better glucose regulation and satiety, and time-restricted eating and intermittent fasting add tools on top of that, especially when they lean toward earlier eating windows rather than later ones. For patients, these strategies are often more about changing the clock than drastically changing the plate.

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

References:

  • Gill S, Panda S. A Smartphone App Reveals Erratic Diurnal Eating Patterns in Humans that Can Be Modulated for Health Benefits. Cell Metabolism. 2015;22(5):789-798. PMID 26411343. https://pubmed.ncbi.nlm.nih.gov/26411343/
  • Jakubowicz D, et al. High Caloric Intake at Breakfast vs. Dinner Differentially Influences Weight Loss of Overweight and Obese Women. Obesity (Silver Spring). 2013;21(12):2504-2512. PMID 23512957. https://pubmed.ncbi.nlm.nih.gov/23512957/
  • St-Onge MP, et al. Meal Timing and Frequency: Implications for Cardiovascular Disease Prevention. Circulation. 2017;135:e96-e121. PMID 28137935. https://pubmed.ncbi.nlm.nih.gov/28137935/
  • Ravussin E, et al. Early Time-Restricted Feeding Reduces Appetite and Increases Fat Oxidation But Does Not Affect Energy Expenditure in Humans. Obesity (Silver Spring). 2019;27(8):1244-1254. PMID 31339000. https://pubmed.ncbi.nlm.nih.gov/31339000/
  • Trepanowski JF, et al. Effect of Alternate-Day Fasting on Weight Loss, Weight Maintenance, and Cardioprotection Among Metabolically Healthy Obese Adults. JAMA Intern Med. 2017;177(7):930-938. PMID 28459931. https://pubmed.ncbi.nlm.nih.gov/28459931/

Does Food Order Affect Blood Sugar? What to Eat First

On By Dr. Scott RennieIn Weight Loss & Metabolic HealthLeave a comment

One of the more practical strategies to come out of recent nutrition research is food order, also called nutrient sequencing. The order in which you eat macronutrients changes how your body responds to the meal, and starting with protein, fat, or fiber before carbohydrate can blunt the post-meal glucose spike and soften the insulin response that follows it.

In a small, tightly controlled study, Shukla and colleagues found that eating vegetables and protein before carbohydrate produced a 73 percent reduction in post-meal glucose and nearly a 50 percent reduction in insulin, compared with eating carbohydrate first (Shukla et al., 2015). Touhamy and colleagues reported something similar in patients with type 2 diabetes: when carbohydrates came last, glucose peaks were 44 percent lower and glycemic variability improved (Touhamy et al., 2025).

People with obesity often report better satiety eating this way. Those with prediabetes see improved tolerance and reductions in HbA1c. Children with type 1 diabetes show smaller post-meal glucose rises when protein and fat come before carbs (Faber et al., 2018). Women with gestational diabetes also see better glycemic control with a carbohydrate-last pattern during pregnancy (Murugesan et al., 2024).

Protein and fat slow gastric emptying. That delays carbohydrate absorption and keeps the glucose peak lower. Incretin hormones like GLP-1 and GIP get stimulated too, which improves insulin sensitivity and overall glucose regulation.

It usually comes down to small shifts: a salad or a non-starchy vegetable first, a few bites of protein, a small portion of healthy fat, and only then rice or bread. Even in food cultures where carbohydrate traditionally opens the meal, reordering it is usually possible. I’ve seen patients make it work by adding a quick vegetable starter at home or choosing a protein appetizer when dining out. They often say it feels more doable than committing to a strict low-carb plan.

No food groups to cut. No restrictive plan to follow. Just change the order in which you eat, and the metabolic payoff can be substantial.

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources:

  • Shukla AP, Iliescu RG, Thomas CE, Aronne LJ. Food Order Has a Significant Impact on Postprandial Glucose and Insulin Levels. Diabetes Care. 2015;38(7):e98-e99. PMID 26106234. https://pubmed.ncbi.nlm.nih.gov/26106234/
  • Touhamy S, et al. Carbohydrates-Last Food Order Improves Time in Range and Reduces Glycemic Variability. Diabetes Care. 2025;48(2):e15-e16. PMID 39688621. https://pubmed.ncbi.nlm.nih.gov/39688621/
  • Faber EM, van Kampen PM, Clement-de Boers A, Houdijk ECAM, van der Kaay DCM. The Influence of Food Order on Postprandial Glucose Levels in Children with Type 1 Diabetes. Pediatric Diabetes. 2018;19(4):809-815. PMID 29527759. https://pubmed.ncbi.nlm.nih.gov/29527759/
  • Murugesan R, Kumar J, Thiruselvam S, et al. Food Order Affects Blood Glucose and Insulin Levels in Women with Gestational Diabetes. Frontiers in Nutrition. 2024;11:1512231. PMID 39777075. https://pubmed.ncbi.nlm.nih.gov/39777075/

Measles in 2025: What Patients and Providers Need to Know

On By Dr. Scott RennieIn Infectious DiseaseLeave a comment

The 2025 measles outbreak was one of the most concerning we’d seen in more than a decade. As of March 2025, Texas alone had reported almost 200 confirmed cases, the highest in years. Other states, including New Mexico, Georgia, Kentucky, New Jersey, New York, Pennsylvania, Rhode Island, Washington, California, and Florida, were seeing rising numbers too. Most of those infected were younger than 19, and 95 percent had either not been vaccinated or had an unknown vaccine status. Roughly one in five patients had needed hospitalization by that point. Tragically, one unvaccinated child in Texas had died.

Lower vaccination rates in some communities made it easier for the virus to spread at the time. Clinicians need to stay alert regardless: a cough, fever, and rash in an area with known cases should still raise immediate suspicion for measles.

What Measles Is

Measles is caused by the measles virus, part of the Morbillivirus family. It’s a single-stranded RNA virus that first infects the respiratory tract, then spreads throughout the body.

How It Spreads

The virus is extremely contagious. When someone coughs or sneezes, droplets can hang in the air for up to two hours, and touching a contaminated surface, then your eyes, nose, or mouth, can spread it just as easily.

If someone is exposed and not immune, nine out of ten will get sick. People with measles can spread the virus four days before the rash starts and four days after. That’s one reason outbreaks move so quickly.

The infectiousness of a virus is often measured by R0. For measles, in an unvaccinated community, R0 runs between 12 and 18, meaning one person can spread it to 12 to 18 others, a transmission rate high enough that public health teams treat a single confirmed case as urgent rather than waiting to see if it spreads further. Get 95 percent of a community vaccinated and R0 drops to about 1, which usually stops an outbreak in its tracks.

Symptoms

The illness follows a predictable pattern. Fever first. Then the rash.

About 7 to 21 days after exposure, early symptoms show up: high fever, cough, runny nose, red and watery eyes. Two or three days later, tiny white spots called Koplik spots can appear inside the mouth. By days 3 to 5 of illness, a red rash spreads from the face and hairline down the body, sometimes raised in the center, often with the fever spiking again at the same time.

Why It’s Serious

Complications are common. Pneumonia is the leading cause of death from measles. About one in a thousand people develops encephalitis, or brain inflammation, which can cause seizures or permanent damage. Severe diarrhea can cause dehydration. In rare cases, blindness or hearing loss occur.

There’s also a delayed complication called subacute sclerosing panencephalitis, a chronic infection of the central nervous system that shows up 6 to 8 years after measles. Symptoms include weakness, tremors, difficulty walking, and eventually coma. It is progressive and untreatable.

Infants, pregnant patients, and people with weak immune systems are at highest risk.

Diagnosis and Treatment

Diagnosis is usually confirmed by blood testing for IgM antibodies, which indicate recent infection. Respiratory swabs from the nose and throat can also detect measles RNA by PCR testing. Sometimes urine is tested as well.

There is no direct antiviral therapy. Treatment is supportive: keeping patients hydrated, managing fever and discomfort with acetaminophen or ibuprofen, and in children, supplementing with vitamin A. The virus lowers vitamin A levels in the body, and supplementing helps reduce the risk of severe complications.

What To Do if Someone Gets Measles

Isolation is critical. A patient should stay isolated for at least four days after the rash appears. Family and close contacts should be alerted. Post-exposure prophylaxis is an option for people who are unvaccinated or whose immunity is uncertain. The MMR vaccine may help if given within 72 hours of exposure. If that’s not possible, immune globulin (IVIG) within 6 days is another option, though the two should not be given together.

Complications need close monitoring. Breathing trouble, high fever that does not improve, or neurological symptoms should trigger immediate medical evaluation.

Prevention

The MMR vaccine is the most effective protection we have. Two doses run about 97 percent effective. Children should get their first dose at 12 to 15 months and the second between ages 4 and 6. Adults without immunity should receive at least one dose, and during outbreaks, infants as young as 6 months may be vaccinated early.

Vaccination protects the person who gets it. It also protects those who can’t get vaccinated themselves, including babies too young for the series and patients with immune conditions that rule out live vaccines. Stopping an outbreak for good takes at least 95 percent community coverage.

Closing

The 2025 measles outbreak showed how quickly this virus can return when vaccination coverage drops. Recognizing symptoms early, isolating cases, and vaccinating remain the keys to controlling it. As healthcare providers, we need to keep talking with patients about the value of the MMR vaccine and stay proactive about reporting and diagnosing cases.

For more information, visit the CDC’s measles page.

—

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources

Tuberculosis in 2025: Kansas Outbreak, Vaccines, and Staying Safe

On By Dr. Scott RennieIn Infectious DiseaseLeave a comment

Tuberculosis is still very much with us. Kansas dealt with a large outbreak in 2025, a reminder of how persistent this disease can be. As of January 2025, more than 67 active cases had been confirmed, mostly in Wyandotte and Johnson counties. There were fatalities, and the number of latent infections was rising at the time. Public health teams worked hard to contain it. It’s a reminder that TB remains a threat in both high-risk groups and the general population.

What TB Is

TB is caused by Mycobacterium tuberculosis. It usually attacks the lungs, but it doesn’t stop there: it can affect the brain, kidneys, spine, and other organs. It spreads through the air when someone with active TB coughs, sneezes, or even talks, and people nearby can breathe in the bacteria without realizing it.

Symptoms and Progression

The first signs can be subtle. A cough that won’t go away, maybe three weeks or more. Some patients cough up blood or sputum. Chest pain, fatigue, fever, night sweats, loss of appetite, and unexplained weight loss are common. Left untreated, TB can destroy lung tissue and spread further, causing meningitis, bone infection, or failure of multiple organs.

Latent vs Active

It’s important to separate latent TB from active disease. Latent TB means the bacteria are present but not causing illness. The person has no symptoms and is not contagious. But the bacteria can “wake up,” especially if the immune system gets weaker, and turn into active TB.

Active TB means the bacteria are multiplying, symptoms are present, and the patient can spread it to others. That’s the form that requires urgent treatment.

How It’s Detected

Testing matters. The skin test (the PPD or tuberculin test) is still used. A small injection under the skin, and the site is checked in 48 to 72 hours. Blood tests like QuantiFERON-TB Gold or T-SPOT.TB are often preferred, especially for people who got the BCG vaccine, since it can interfere with skin test results.

If TB is suspected, chest imaging can show lung changes, and sputum culture confirms the presence of M. tuberculosis. That culture is the gold standard for diagnosis in someone with symptoms.

The Role of the BCG Vaccine

The only vaccine we have is BCG. It’s mostly given to infants in countries where TB is widespread. It helps protect children from the most severe forms of TB, like meningitis, but it’s less effective at preventing pulmonary TB in adults. In the United States, it’s not routinely used because of its limited impact on adult disease and the way it interferes with skin testing.

Some healthcare workers in high-risk settings or people with repeated exposure may still receive it.

Protecting Against TB

Vaccination isn’t the whole answer for TB. Early detection and treatment carry just as much weight, maybe more. People at higher risk should be screened regularly, and treating latent TB is critical, because it stops progression to active disease.

Simple steps help too: masks in healthcare settings, good airflow in crowded places, covering your mouth when you cough, washing your hands regularly. During outbreaks, N95 or P100 respirators are the masks that actually filter TB bacteria from the air. Surgical masks don’t provide the same protection.

Treatment and Why It Has to Be Completed

TB can be treated, but it takes time. Standard therapy is multiple antibiotics for six to nine months. Isoniazid, rifampin, ethambutol, and pyrazinamide are the most commonly used.

Stopping treatment too soon is dangerous, because that’s exactly how drug-resistant TB develops, and multidrug-resistant TB is much harder to treat, far more expensive, and comes with worse outcomes across the board. An incompletely treated patient is also still contagious. Public health departments often use directly observed therapy, or DOT, where someone watches patients take their medications. It might sound strict, but it works. It prevents resistance and saves lives.

Public Health and What’s Next

TB is a problem here at home, and the outbreak in Kansas proves it. It spreads any time we let our guard down. Healthcare providers need to push for routine testing in high-risk groups, make sure patients complete treatment, and support public health efforts.

Patients and communities have a role too: stay informed, recognize symptoms, seek evaluation early. And we need continued research into better vaccines and treatments.

—

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources

Man wearing protective mask with smoky industrial city background

LA Wildfires: Health Risks and a Doctor’s Perspective

On By Dr. Scott RennieIn Travel, Wilderness & Preparedness MedicineLeave a comment

On January 7, 2025, powerful winds sparked wildfires across Los Angeles. I live only a few blocks from one of the burn zones, and like many in the city, I was trying to keep up with the immediate challenges while also absorbing what it meant for health and safety. Those fires were a reminder of just how fragile our environment and infrastructure can be when nature takes over.

The air was heavy with smoke and ash in those first weeks. Step outside and you could taste it, feel it in your throat. Air quality monitors showed “unhealthy” or “very unhealthy” levels for days, mainly due to fine particulate matter, PM2.5. Even indoors, it was hard to escape. The smell crept through closed windows and doors. I’ve had patients call worried about worsening asthma, chest tightness, or headaches after only short exposures.

The winds drove the flames and knocked out power across neighborhoods. My own area went days without electricity, which made it harder to stay updated on evacuation alerts or even preserve basic supplies. There was concern about the water too. Ash and debris can wash into reservoirs and pipes, and that raised real questions about whether tap water was safe to drink. Many of us turned to bottled water, but the shelves emptied fast.

In those weeks, each day brought news of flare-ups in other parts of the region. Containment lines held in one spot, only to break in another. Watching the wind shift kept you tense. We’d all packed bags and set them by the door, not knowing when an evacuation order might come.

Ash settled over yards, on car hoods, across rooftops. The instinct is to clean it as fast as possible, but that can cause more harm than good: leaf blowers just push particles back into the air, where neighbors and children breathe them in. A safer approach is low-tech: a vacuum indoors, or a wet mop and gentle sweeping outside. When I cleaned my porch, I wore an N95 mask, gloves, and goggles. Ash went into sealed trash bags so it wouldn’t scatter again.

Protecting yourself during a wildfire comes down to layers of defense. Stay indoors if the air is poor. Use HEPA filters if you have them. Step outside only if you need to, and wear an N95 mask against fine particles. Skip the outdoor exercise. For water, use bottled supplies when you can, or follow boil-water notices if officials issue them. Keep a go-bag ready with medications, documents, food, and water, and know your evacuation routes and how you’ll get updates, whether that’s a radio, phone alerts, or a neighbor who checks in.

These fires took a toll on health, both physical and mental. But I saw resilience too. A neighbor came by to offer us extra bottled water when supplies ran low. Others opened their doors to families who had to evacuate. The community response was one of the few bright points in a difficult stretch.

It’s clear that Los Angeles needs to strengthen preparedness, both individually and collectively. Climate change is making fire seasons longer and more intense. Infrastructure will have to adapt, but so will we as individuals. Preparedness, whether that’s sealing a window with weather stripping, having an air purifier ready, or knowing your evacuation plan, can make a real difference.

At the time, the focus was on protecting ourselves and helping each other through it. Nobody knew how many more weeks the fires would burn, but people in LA were resourceful, and determined to get through it together.

—

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources

Flu Prevention: Vaccines, Symptoms, and Treatment Options

On By Dr. Scott RennieIn Infectious DiseaseLeave a comment

Influenza causes major illness every year despite vaccines and treatment options. The 2024-2025 season was a busy one. By mid-January 2025, the CDC had reported over 10 million flu-related illnesses and about 120,000 hospitalizations, numbers that ran ahead of the prior year. Flu season typically peaks between December and February. That swing from year to year reminds us how much flu activity depends on strain changes, how many people get vaccinated, and what immunity looks like across the population.

Why History Matters

Flu is not new. The 1918-1919 pandemic killed an estimated 50 million people worldwide. Three later pandemics, in 1957, 1968, and 2009, each showed how dangerous a new strain can be. Every one of them pushed public health toward stronger vaccines, better antivirals, and better surveillance.

Public Health Impact

Every year influenza causes between 290,000 and 650,000 deaths worldwide. Young children, older adults, pregnant people, and those with chronic conditions carry the most risk. Flu season also strains hospitals and clinics, and it costs billions in missed workdays and healthcare spending on top of the direct health toll.

How Flu Spreads

The incubation period is short, usually about two days. Transmission happens through droplets, when people cough, sneeze, or talk, and through contaminated surfaces if someone touches their face afterward. In households and schools, attack rates can reach 20 to 30 percent.

People can spread flu a full day before symptoms start, and for up to a week after. Immunocompromised patients can spread it longer than that.

Symptoms and Complications

Classic symptoms are fever, cough, sore throat, body aches, fatigue, and headache. Children are more likely than adults to get vomiting or diarrhea along with it.

For most people the illness runs its course. But complications are common enough to take seriously: secondary bacterial pneumonia, worsening asthma or COPD, myocarditis, encephalitis, even ARDS. These are the cases that fill hospital beds every winter.

Influenza A and B

Both influenza A and B drive seasonal flu, but they behave differently. Influenza A is more common, infects humans and animals, and is behind pandemics. Subtypes like H1N1 and H3N2 are defined by their surface proteins. Influenza B only infects humans, and it has two main lineages, Victoria and Yamagata. Outbreaks from influenza B tend to be smaller but still cause serious illness, especially in children. A often dominates earlier in the season, while B shows up later, though they can circulate together.

Diagnosis

Most of the time, diagnosis starts clinically. But testing can confirm it. Rapid tests give results in about 15 minutes, though sensitivity is limited. RT-PCR is much more accurate and is considered the gold standard.

Vaccination

The flu vaccine changes yearly to match expected strains. For 2024-2025, the U.S. vaccine was trivalent, not quadrivalent: two influenza A strains, H1N1 and H3N2, and one influenza B strain, Victoria. B/Yamagata was left out of that season’s formulation, per the CDC’s ACIP recommendations, because global surveillance had not detected it since 2020.

Effectiveness runs 40 to 60 percent, depending on the match and the patient’s age and health. Vaccines come in several forms: inactivated injectable, live attenuated nasal spray, and higher-dose or adjuvanted versions for older adults.

Treatment

Antivirals work best started within 48 hours of symptoms. Oseltamivir is oral and the one used most. Zanamivir is inhaled, peramivir is intravenous and usually reserved for hospitalized patients, and baloxavir is a single-dose oral option that blocks replication.

Supportive care still matters most: rest, fluids, acetaminophen or ibuprofen for fever and pain, and close monitoring for complications in anyone at higher risk.

Prevention

Annual vaccination is the best prevention. Handwashing, covering coughs, staying home when sick, and avoiding close contact with ill people all reduce spread. Masks and improved ventilation are useful in crowded settings, especially when flu activity runs high.

Final Notes

Flu is not going away. Each season differs, but the tools we have (vaccines, antivirals, good hygiene, early recognition) make a real difference when used consistently. As physicians, we need to push vaccination, treat high-risk patients early, and keep reminding our communities that influenza is more than “just a cold.”

—

Scott Rennie, D.O.
Board Certified in Obesity Medicine and Family Medicine

This blog is for educational purposes only and does not constitute individual medical advice. Always consult your own physician before making changes to your health, medications, or treatment plan.

Sources

  • Centers for Disease Control and Prevention. FluView Surveillance. https://www.cdc.gov/fluview/surveillance/index.html
  • World Health Organization. Influenza (Seasonal). https://www.who.int/news-room/fact-sheets/detail/influenza-(seasonal))
  • Dawood FS, et al. Estimated global mortality associated with the first 12 months of 2009 pandemic influenza A H1N1 virus circulation: a modelling study. Lancet Infect Dis. 2012;12(9):687-695. PMID 22738893.
  • Grohskopf LA, et al. Prevention and Control of Seasonal Influenza with Vaccines: Recommendations of the Advisory Committee on Immunization Practices, United States, 2024-25 Influenza Season. MMWR Recomm Rep. 2024;73(RR-5):1-25. PMID 39197095.